Is COVID-19 Caused by a Virus or a Mycobacterium?

“It can boost the immune system so that it defends better against a whole range of different infections, a whole range of different viruses and bacteria in a lot more generalized way,” said Curtis, who’s also a professor of pediatric infectious diseases at the University of Melbourne and head of the infectious diseases unit at the city’s Royal Children’s Hospital.

And that is exactly how BCG works  ̶  not against "a virus" and not through “training” or “bolstering” the immune system, but by BCG (Mycobacterium bovis) shooting off phages to kill closely related mycobacteria or mycobacterial colonization, latent or active, in the system. Since phages are species specific, they are often used for diagnostic purposes. So just the mere successful use of dilute mycobacteria (BCG) with its mycobacteriophages is evidence in itself that the target, the coronavirus, is not of a spontaneous occurrence, but a mycobacterial generated disease."

Quote:

So just the mere successful use of dilute mycobacteria (BCG) with its mycobacteriophages is evidence in itself that the target, the coronavirus, is not of a spontaneous occurrence, but a mycobacterial generated disease.

Can you explain to your readers how the BCG vaccine would bolster/improve the immune system so as to eradicate SARS-Cov-2? If you cannot, please write to Dr. Curtis and ask him to do it. What is the specific mechanism by which mycobacterium bovis attacks a virus, through the immune system?

Don't worry, you won't find a single paper which can explain this.

I have already provided the source: peer-reviewed, Pulmonary Research and Respiratory Care journal.
https://www.academia.edu/42379130/IN_PRESS_PROMISING_ANTIMICROBIAL_HOPE_FOR_CORONAVIRUS_BUT_IS_IT_WORKING_AGAINST_A_VIRUS_......................................................................Dr._Lawrence_Broxmeyer_MD

Very simple mechanism, but it works ONLY for mycobacterium.

Sure.

Can you explain how the "immune defense" will fight an infection using BCG, other than the mycobacteriophages mechanism?

Specifically, how would SARS-Cov-2 be eradicated using BCG (of all possible things)?

In the case of mycobacterium, things are very simple, the photographs provided earlier even capture the very moment the mycobacteriophages attack mycobacterial colonization.

How would a virus be influenced, via the immune system, by BCG?

Here is a second reference, this time published in the most prestigious journal in the world on infectious diseases:

Broxmeyer, L., Sosnowska, D., Miltner, E., Chacon, O., Wagner, D., McGarvey, J., Barletta, R.G. and Bermuddez, L.E. (2002) Killing of Mycobacterium avium and Mycobacterium tuberculosis by a mycobacteriophage delivered by a nonvirulent mycobacterium: a model for phage therapy of intracellular bacterial pathogens. J Infect Dis 186,
1155–1160

https://academic.oup.com/jid/article/186/8/1155/2191390

The Journal of Infectious Diseases
Oxford Academic

Do you recognize the first name in the authors' section?

Yes, it is Dr. Lawrence Broxmeyer himself.

Actual quotes from sokarul:

"You have to get over the fact that two things can be equal and not be the same thing.

A dead particle does not equal an alive particle.

It it theories water came from asteroids.

So the ground accelerates them, then why do they not leave the ground?

I wasn't thinking about the other type of acceleration."

"You have to get over the fact that two things can be equal and not be the same thing.

A dead particle does not equal an alive particle.

It it theories water came from asteroids.

So the ground accelerates them, then why do they not leave the ground?

I wasn't thinking about the other type of acceleration."

Broxmeyer, L., Sosnowska, D., Miltner, E., Chacon, O., Wagner, D., McGarvey, J., Barletta, R.G. and Bermuddez, L.E. (2002) Killing of Mycobacterium avium and Mycobacterium tuberculosis by a mycobacteriophage delivered by a nonvirulent mycobacterium: a model for phage therapy of intracellular bacterial pathogens. J Infect Dis 186,
1155–1160

https://academic.oup.com/jid/article/186/8/1155/2191390

The Journal of Infectious Diseases
Oxford Academic


The first quoted user, sokarul, calls Dr. Lawrence Broxmeyer, who was peer reviewed and was able to publish his research in the most prestigious infectious diseases journal in the world, a clown. How does this work?

Once again it is explained by one of the doctors you quoted.

But it isn't, for if it was, you'd have been able to quote the specific passages where we learn how BCG improves the immune system so as to stop SARS-Cov-2.


In the case of mycobacterium, there is a very simple and direct explanation.

For SARS-Cov-2, there is none.

Even news reporters know the answer.

Sure. Then, ask the reporter how does he/she know this fact. The answer, invariably, will be to direct you to the top epidemiologist in their respective country. Now, ask that epidemiologist how he/she had arrived at this wondrous conclusion. They will say "RNA testing", "passing through microfilters". Now, remind them that mycobacterium also have these features.

I have already done my homework on this subject. Rest assuredly that there is no specific answer coming from Dr. Curtis, he cannot offer any other than a vague response such as "improve the immune system".

"In nature there is a phenomena which happens probably millions of times a day in which one colony of bacteria or mycobacteria sends out its viral phages (bacteriophages or mycobacteriophages) that live inside it to kill another colony of the same type of organism......a sort of natural lysogeny. The bacterialviruses called phages are the most plentiful viruses on earth. But they are species specific in that they only attack like species. The Mycobacterium bovis in BCG are closely related to and in fact part of the Mycobacterium tuberculosis complex. Therefore, the phages inside BCG have the potential to destroy TB and other related mycobacteria.

The destruction of TB bacilli in the body is in and of itself “bolstering the immune system”, as there is no other microbe known to man that is quite as immunosuppressive.

And that is exactly how BCG works  ̶  not against "a virus" and not through “training” or “bolstering” the immune system, but by BCG (Mycobacterium bovis) shooting off phages to kill closely related mycobacteria or mycobacterial colonization, latent or active, in the system. Since phages are species specific, they are often used for diagnostic purposes. So just the mere successful use of dilute mycobacteria (BCG) with its mycobacteriophages is evidence in itself that the target, the coronavirus, is not of a spontaneous occurrence, but a mycobacterial generated disease.[.b]"





This is what BCG does for mycobacterium.

Go ahead, and find the similar mechanism for a virus, specifically SARS-Cov-2.

You found two articles.

No. The statement that I did my homework refers to the fact that I have thoroughly researched your best case scenario (BCG to SARS-Cov-2 connection).

No. It is the most direct mechanism, via mycobacteriophages, of transmission from BCG to mycobacterium. The mechanism has been captured on film.

Go ahead and find a similar mechanism for SARS-Cov-2. You won't find any. Not even a conjecture.

That would be very bad news for your side.

If BCG treats only the symptoms, then you have this to deal with.

Mycobacteria tuberculosis (TB) acts like a virus. It is a seasonal infection peaking in winter just like cold and flu viruses. While the TB mycobacterium is spread throughout the year, it is only when vitamin D levels are low and the immune system weak that it produces symptoms. It is a cell-wall deficient germ that appears like a virus under a microscope. It attacks the lungs, resulting in inflammation that essentially drowns infected patients who cannot breathe, just like COVID-19 coronavirus is said to do.

How, then, does Dr. Curtis know that it is the SARS-Cov-2 which caused the infectious outbreak? If it doesn't have to attack the virus, how then does he know what is the primary cause? Mycobacterium also has RNA, and passes through microfilters.

If it is only the symptoms that are going to be treated, then the connection between these symptoms and mycobacterium are much more direct and logical, than between the common cold (coronavirus) and the clinical manifestations of this illness.

Moreover, he still lacks the mechanism by which the treatment of these symptoms by BCG will cure the virus itself.

For mycobacterium the mechanism is very simple, direct and has been proven to exist.

Even news reporters know the answer.
Sure. Then, ask the reporter how does he/she know this fact. The answer, invariably, will be to direct you to the top epidemiologist in their respective country. Now, ask that epidemiologist how he/she had arrived at this wondrous conclusion. They will say "RNA testing", "passing through microfilters". Now, remind them that mycobacterium also have these features.

Cheap blood test can discriminate between bacterial, viral infections
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The overuse of antibiotics threatens to create a global scourge of antibiotic-resistant bacterial pathogens. Because of this problem, public health experts regularly remind physicians to prescribe antibiotics only for bacterial infections.
But too often there’s no easy way for doctors to tell whether a patient’s illness is bacterial or viral or, sometimes, if there’s any infection at all.
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Blood test
The team used publicly available patient gene expression data to pinpoint just seven human genes whose activity changes during an infection; their pattern of activity can distinguish whether an infection is bacterial or viral.

When pathogens infect the cells of the body, the infection sets off a chain reaction involving the immune system that changes the activity, or expression, of hundreds of genes. Gene expression is the process by which cells extract information from genes and render it in the form of either proteins or RNA. Cells have the capacity to express more or less of each molecule, creating a pattern of gene expression that changes in response to external influences, including infections.

The seven-gene test is a vast improvement over earlier tests that look at the activity of hundreds of genes, the researchers said. Because so few genes are involved, the new test will be cheaper and faster, while remaining accurate, they said.

Ask your source Dr. Nigel Curtis.
I have already done my homework on this subject.

Rest assuredly that there is no specific answer coming from Dr. Curtis, he cannot offer any other than a vague response such as "improve the immune system".

"In nature there is a phenomena which happens probably millions of times a day in which one colony of bacteria or mycobacteria sends out its viral phages^[1] (bacteriophages or mycobacteriophages) that live inside it to kill another colony of the same type of organism......a sort of natural lysogeny. The bacterialviruses called phages are the most plentiful viruses on earth. But they are species specific in that they only attack like species. The Mycobacterium bovis in BCG are closely related to and in fact part of the Mycobacterium tuberculosis complex. Therefore, the phages inside BCG have the potential to destroy TB and other related mycobacteria.

The destruction of TB bacilli in the body is in and of itself “bolstering the immune system”, as there is no other microbe known to man that is quite as immunosuppressive." by Dr. Lawrence Broxmeyer, MD

Go ahead, and find the similar mechanism for a virus, specifically SARS-Cov-2.

and virii do not have DNA.

COVID-2 is caused by the Mycobacterium tuberculosis

and virii do not have DNA.

Should I quote the moron who claimed the COVID-2...

All viruses carry DNA.

All RNA viruses carry DNA.

Further information:

RNA Viruses

RNA viruses, also known as retroviruses, have RNA as their genetic material. Some examples of retroviruses are hepatitis viruses and HIV. When these viruses enter a host cell, they must first convert their RNA into DNA. This process, called reverse transcription, enables the virus to inject its genetic material into the host cell and use the host's biochemical machinery, similar to a DNA virus.

Often, retroviruses use an enzyme, called integrase, to insert the retroviral DNA into the genome of the host cell (the ability of retroviruses to integrate this DNA into the host cell’s DNA).


More information:

https://www.verywellhealth.com/hiv-is-a-retrovirus-what-does-that-mean-3132822

Usually, cells convert DNA into RNA so that it can be made into proteins. But with retroviruses, the process has to start by going backward. First, the viral RNA is transformed into DNA. Then the cell can copy the DNA. The cell can also transcribe the DNA back into RNA as the first step in making viral proteins.


All viruses carry DNA.